HOW TO PRESCRIBE PRISM GLASSES

 Prescribing Prism

Both phorias and tropias can be accurately measured using prism and cover tests. Initially a cover test is performed to determine the fixing eye and estimate the deviation. This is followed by the prism and alternating cover test, with adjustment of prism strength until refixation movement of the eyes is neutralized.

Phorias

Although most normal individuals have at least some phoria, the vast majority are asymptomatic. If a deviation is uncovered on routine testing in an asymptomatic patient, no treatment is necessary. If a patient is complaining of asthenopia and a deviation is discovered during testing, one must first ensure that there are no coexisting issues prior to attributing the symptoms to a phoria. There are a number of causes for asthenopic symptoms that should be ruled out. Some of these causes are summarized in Table 1. To be sure, some of the refractive causes of asthenopia are via induced phorias.

Once other causes for symptoms are ruled out, phorias should be investigated, and, in certain cases, may benefit from partial prism correction. One indication that the patient will benefit from prisms is minimal prism adaptation during a 15- to 20-minute trial with prisms in the waiting area. Otherwise, if there is a significant adaptation to the prisms, the patient’s symptoms will probably not be improved through the use of prisms, and other therapies, including surgery, should be considered.

Esophoria

As soon as the cover/uncover test identifies an esophoria, the next step is to obtain an accurate cycloplegic refraction because many cases of esophoria are “accommodative,” due to uncorrected or undercorrected hyperopia. It is wise to perform the cover/uncover test for esophoria at both distance and near, for the deviation may be larger at one distance than the other.

After refracting the patient and ensuring that there is no uncorrected hyperopia, one may wish to try a small amount of plus sphere to decrease the accommodative demand, as some cases of esophoria are due to accommodative excess. Such intervention is often enough to treat the condition, but base-out prism may be necessary if refractive methods fail. In situations that require the use of prisms, base-out prism should be prescribed with only the minimum amount of power required to eliminate the symptoms.

Exophoria

Again, careful refraction of the patient can help the management of many cases of exophoria. With refractive correction in place, if any, cover tests should be performed, and accommodation should be evaluated by push-up measurement of accommodative amplitude, or, especially in children, by dynamic retinoscopy. Divergence excess (in contrast to convergence insufficiency) manifests as an increased angle of exophoria in the distance.

In exophoria, correcting both myopia and hyperopia can help improve symptoms, but additional cautions should be taken when correcting hyperopia, as full correction of hyperopia may worsen the symptoms. A several-minute test with hyperopic correction should be attempted to see if exophoric symptoms improve because of clearer imagery or worsen by relaxing accommodation. If they worsen, prescribe the largest correction possible to treat the hyperopia while avoiding exophoric symptoms. A good starting point is one-third of the spherical error. Just as plus lenses can be helpful for esophoria, decreased-power plus lenses or even minus lenses can improve exophoria.

Base-in prisms may also be helpful for the treatment of exophoria. As with esophoria, the least amount of prism that eliminates exophoric symptoms should be used. The cover test should provide an estimate of the power of the prism to be used. Additionally, treating a minor hyperphoria with vertical prisms can allow the patient to compensate for exophoria with no need for horizontal prisms (see below). If divergence excess is found to be the cause of the exophoria, prisms should be avoided. In this situation, base-in prism can cause esophoria at near, which patients do not tolerate well.

Hyperphoria

Both the measurement and treatment for hyperphoria are similar to those for the horizontal heterophorias. One important point regarding hyperphorias is that they often coexist with horizontal heterophorias, and the treatment of one may improve or eliminate the other condition. Therefore if either a horizontal heterophoria or a vertical heterophoria is found, it is important to investigate whether the other is present. Initial attempts at treatment should be focused on the primary phoria with the intention of treating both.

Tropias

various types of tropias can be categorized in a number of ways. Broadly, they can be considered as comitant or incomitant. Surgery is generally the preferred treatment for tropias unless there is a reason not to perform surgery (see above). Therefore prisms for tropias are generally used as a temporizing measure until surgery can be performed.

In general, the measurement and treatment of tropias parallel those of phorias, as discussed above. A 30- to 45-minute patch test can be especially useful in uncovering the full deviation. When measuring incomitant deviations with prisms with each eye “fixing,” it is imperative to switch the prism to always be before the non-fixing eye. Otherwise the “fixing” eye behind the prism will not truly be looking in the intended direction, and major measurement errors can occur. For comitant strabismus, a prism adaptation test can be helpful to help determine treatment. For incomitant strabismus, prisms may be helpful to move an area of single vision to straight ahead.

Anisometropia

Often when patients with anisometropia receive a new pair of glasses, they will complain of double vision, particularly while reading. This double vision is due to the differential prismatic effects of the two lenses when the patient is looking off-center as when reading (as per Prentice’s Rule). In order to improve reading vision, vertical prism can be incorporated into the lower portion of one lens or the other to help compensate for the differential vertical prismatic effect and lessen or eliminate the double vision. These prisms are referred to as slab-off or reverse-slab prisms. The slab-off prism is placed on the more minus or less plus lens, more commonly on glass lenses, and in effect takes away base-down prism (adds base-up prism). The reverse-slab prism is placed on the more plus or less minus lens, most commonly on molded plastic lenses, and adds base-down prism.

Rather than a calculation of the amount of slab-off or reverse-slab prism to prescribe, trial-and-error measurements are preferred because one does not know how much the patient has already compensated to previous anisometropic glasses. Increasing the amount of prism handheld over the lower portion of one lens of the anisometropic correction until the patient can read comfortably is the most reliable way to determine the amount of slab-off or reverse-slab prism to prescribe. Up to 4 to 6 of slab-off or reverse-slab effect can be obtained when needed.

 Pitfalls in Measuring Deviations

When prisms are used to measure a strabismic deviation in a patient, several easily avoidable mistakes are commonly made.

Positioning

The position in which the prism is held can be critical when measuring a patient’s deviation. There are three intended positions for holding prisms: the Prentice position, the minimum deviation position, and the frontal plane position . Glass prisms are calibrated for the Prentice position and should be held in this manner when making measurements. Plastic prisms, including plastic prism bars, are generally calibrated for the minimum deviation position. As it is difficult to estimate accurately the minimum deviation position, the frontal plane position is a good approximation when using plastic prisms.

Adding Prisms

Stacking two prisms in the same direction, especially if one is of high power, can also lead to errors because of the same positioning issues mentioned above. If the two prisms are held in contact with each other, even if the first prism is held in the correct position, the second prism will not be in the correct position in relation to the light leaving the first prism. This will create a stronger prism effect than the sum of the two prisms, leading to a falsely low measurement. If the sum of the two prisms is prescribed, or surgery is based on the prism measurement, the patient will be left undercorrected. It is difficult to estimate accurately the correct position of the second prism in relation to the first, and it is therefore more accurate to simply stack the prisms together and use a conversion table to add their true effects.

Splitting Prisms Between the  Eyes

One might assume that one way to avoid the difficulties of stacking two prisms is to split the prisms between the two eyes and add the powers together. Although this may work for low prism powers, it becomes increasingly inaccurate with increasing prism power. Splitting prisms is preferred over stacking two prisms together in the same direction before one eye, but again using a table of summed prism values is preferred to avoid errors.

Method for Calculating Oblique Prism

There is a simple method to calculate oblique prism from combining a horizontal prism with a vertical prism that does not require trigonometric calculations and requires only a piece of paper, a ruler, a pen or pencil, and the protractor on a phoropter or trial frame.

First, sketch the vector addition of the two prism powers on polar coordinates to determine the approximate angle for the base direction of the oblique prism. Up is base-up, and right is base-in or base-out depending on which eye the prism is intended for.

Then measure off the number of prism diopters of the two component prisms on two adjacent edges of a piece of paper. (One can use any unit for this measurement, but the unit must be consistent. For example, if you have two prisms, one of 3 and the second 2, measure off 3 cm/inches/etc on one edge and 2 cm/inches/etc on the other edge). Connect the two measurement marks with a line, forming a triangle. Now measure the length of the connecting line you just drew, the hypotenuse of the triangle. This will give you the number of prism diopters for your oblique prism.

Then fold the paper along the hypotenuse, identify the acute angle of the triangle that you estimated using your polar coordinate plot, and measure this angle using the protractor on the phoropter or trial frame, giving you the base direction of the oblique prism.

Uncommon but Important Uses for Prisms Childhood Cranial Nerve Palsies (Third, Fourth, and Sixth Cranial Nerves)

For children with cranial nerve palsies, early treatment is important to prevent amblyopia. Prisms may be used for small, reasonably comitant deviations in order to maintain binocular function. A test with temporary plastic Fresnel prisms is recommended prior to grinding prisms into the child’s lenses. Treatment of a recognized underlying cause is essential, and if prism therapy fails, surgery should be considered.

Prisms for Enhancing Communication

For patients with no useful vision in the deviated eye, prisms can be extremely helpful in improving the appearance of the eyes and facilitating the patients’ communication and interaction with others by alleviating difficulties with eye contact. A prism held opposite the direction of a correcting prism can improve the apparent alignment of the eyes (ie, base-in for esotropia and base-out for exotropia). Expect approximately 1 mm of apparent eye shift for every 8 of prism power.

Homonymous hemianopia

Prisms (or mirrors) may be used in patients with homonymous hemianopia to bring images from an area within the visual field defect into the area with retained vision. Although this may be useful on occasion, generally very high prism powers are required, which can create cosmetic problems as well as a confusing visual environment in which objects may appear and disappear from view unexpectedly.

Hemispatial neglect

Patients who experience right hemisphere strokes often experience left hemispatial neglect. Recent studies have shown that yoked prisms which move both visual fields to the opposite side (to the right) improve function in these patients. The mechanism for this improvement is believed to be that in order to compensate for the shifted binocular visual field, the patient must remap his  sensorimotor coordinates leftward, and this has been shown to improve function on the neglected left side.

Nystagmus with head turn

Patients with a head turn to compensate for nystagmus can benefit from yoked prisms, but just as with patients with homonymous hemianopia, they will often require very high prism powers which may lead to decrease in visual acuity (especially with Fresnel prisms), chromatic aberration, heavy lenses, and cosmetic problems, as well as the visual disturbances described above. In spite of these problems, prescription of bilateral yoked prisms, with base in the same direction as the head turn, can keep the patient’s eyes in an eccentric null position while lessening the head turn.

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The visual pathway

 The visual pathway so we're going to take how the light hits the retina and we're going to take it through the optic nerve and all the other different process if you guys have already watched our video on the phototransduction cascade that's going to be very very important that you do that before we get into this visual pathway because we already talked about exactly how those light rates got converted into chemical changes and then electrical changes and how they went down the axon to the ganglion cells which basically made up the optic nerve all right so now we're going to have two eyeballs right


this is the we're going to say this the left eyeball and this is the right eyeball okay now first things first what I want us to try to imagine if we're gonna put a lot of different concepts together when you guys are looking you guys are like looking at anything around you we our eyes are so amazing that they can pick up different types of visual fields so for example this is the right visual field or the right visual field and this is the left eyes visual field okay so what is this one right here this is the right eyes visual field and this one over here is the left eyes visual field okay now with in the visual field you have two different components of this visual field we're going to say this is the nasal so since your you'd have your honker right there pretend this is your nose okay so pretend this structure here is your nose okay since this is the nose these two structures that are near the nasal region this is the nasal component of the visual field alright so this is the nasal component of the visual field and then this one over here would be near the temple so this is the temporal component of the visual field and this over here is going to be the temporal component of the visual field okay now to make it easy though now that we know that this is what it is it's nasal and the temporals these words are going to get interchanged a lot so I do not want to confuse you guys so what I'm going to do is for the sake of it is I'm going to switch nasal and since this is the right visual field this is what gets confusing the right visual field has a left visual field and a right visual field okay so the right eye has a left visual field and a right visual field so we're going to say this is the left visual field and this is the right visual field okay so I just want to make it as simple as I possibly can here this whole right eye is going to have two fields it's going to have the right visual field and last visual field same thing this left eye is going to have two visual fields it's going to have a left visual field and a right visual field okay simple as that now we're going to be able to kind of do this a lot easier now so instead of using the words nasal and tempore but understand those words because it's going to be important lesions okay let's say first off when we're looking out let's say that this part of the retina there's two parts of the retina so this is the right eye this part of the retina is closest to the Temple or the right temple right so we're going to call this part of the retina we're going to call this the temporal Hemi retina okay that's a temporal Hemi retina now here's what gets really funky the temporal Hemi retina is receiving light rays from the left visual field okay so from here this part here this left visual field is hitting this part of the retina okay so if light rays from the left visual field are hitting this part of the hit retina that temporal heavy right now but then the what is this one this one is close to the actual nose so since it's close to the nose this is called the nasal right Hemi retina so it's called the nasal hemorrhage now and it's called the temporal Hemi retina the nasal hemorrhage now is receiving light from the right visual field so the nasal Henry retina is getting hit with light from the right visual field .

RETINA

 The retina 

The retina which is the deepest layer of the eye remember that we have a fibrous tunic which consists of the sclera and cornea then we have the vascular tunic which contains the choroid and then the neural tunic which has a lot of neurons as you can see and that really is just the retina and we'll go to this image real quick just for a minute but when light enters your eye it's gonna go through the pupil obviously and it's gonna go through to the back of the eye and here's the retina and so in doing so in following that path the light's gonna have to pass through the cornea the anterior chamber the posterior chamber the lens and then through the vitreous chamber and then finally that light is gonna strike the retina at the back of the eye in which you can see a little bit more macroscopically in this picture right here and within the retina we have three cell types the first type is called the photoreceptor cells or just photoreceptors and these are the cells that initially detect the light and there are two subtypes of photoreceptor cells and those are rods and cones we're gonna have a separate video where we go over the major differences between these but I'll just say this for now rods detect non colored light so non color vision just bright and dark and cones detect color okay see four cones see four color then we also have bipolar cells and then we have ganglion cells which eventually become continuous with the optic nerve these are the three major cell types there's also a couple other cell types called amacrine cells and horizontal cells and we're not going to get into these very much most Anatomy courses don't but they just exist pretty much to find tune and regulate the functions of these cell types particularly bipolar cells and ganglion cells okay now before we go any further I want you to notice something so we've got light that we mentioned was passing through the eye okay like this in this direction and then it strikes the retina but what's interesting about the setup of this is it actually doesn't encounter the photoreceptors first actually if we follow the passage of light the photoreceptors are at the back of the eye they're there actually we could say superficial to the ganglion cells so actually the light has to travel through the ganglion cells and then through the

bipolar cell layer and then finally is

able to make contact with the

photoreceptors but it's the

photoreceptors that initially detect

that light and then the photoreceptors

will have an effect on the bipolar cells

which will then in turn have effects on

the ganglion cells and we're gonna look

at that now in this slide okay now to

really understand what happens here we

have to understand what's happening in

the dark so imagine a situation where it

is complete darkness okay

so let's first of all say it's night

it's dark out you're in your room

there's no night lights doors are closed

and you have blackout curtains so you

can't see anything all right so I have

the photoreceptor cells in blue now

interestingly in the dark the

photoreceptor cells are actually


depolarized okay so the photoreceptor

cells are actually D polarized and that

not may not now that probably doesn't

make a lot of sense because usually only

think of depolarization we think of

activating but trust me this is how it

is and it will make sense in the end so

in the dark photoreceptor cells rods and

cones are depolarized that being said

that means the photoreceptors are

activated and they activate the next

neuron in sequence which is called a

bipolar cell it's also worth mentioning

that the way that photoreceptors

activate the bipol

Vitreous Humor

 Today I'm going to talk about vitreous

opacities and vitreous floaters so I

wanted to express some gratitude for a

couple people one is Lilian in our

office who has helped us extensively

with our presentations and I'm really

grateful to her for that and this is a

picture of her here and also I'd like to

express gratitude to Geri seabag who is

a virtual retinal surgeon in California

who has been a pioneer in vitreous

opacities and floaters  and not only

is he a a very erudite and sophisticated

person but he is he has been

consistently and politely pushing this

field forward for many decades

particularly given the attitude that

many of us have towards patients with

floaters and dismissing their symptoms

and he's been very gently and

persistently moving the field in the

right direction so first we'll start

with vitreous Anatomy and over here on

on the Left panel you can see a autopsy

eye in which the choroid the sclera was

removed and the choroid was detected

anteriorly and then the vitreous base

was the second off of the retina and you

could see that early in life the

vitreous gel is firm it's very clear and

and it's it has a structure.


It doesn't

just collapse when you set it on a on a

surgical cloth and if we look at the

iron cross section we can see that the

the vitreous emanates from the aura

sirata and it's always connected to this

space during life there's a little space

right here and then the vitreous is

connected to the lens and this space

here behind the lens that posterior

capsule is called Berger space or the

retro mental space of galette

there are fibers that extend up to the

pars plana and even the pars Picatta and

these aren't shown on this diagram but

we know that those exists because when

we're opera

patients with proliferative retinal

diseases we can see contraction with the

retina actually gets pulled from here up

onto the ciliary body and by coming

across with a cutter in shaving those

vitreous bands we can release that

traction

there's cloak haze canal which emanates

from the disk and if we blow the space

up here we can see this this area a

little better there's this little space

here a little space behind the capsule

and then this type adhesion between the

anterior vitreous face and the posterior

capsule and this is why cataract

surgeons get vitreous loss when they

break the posterior capsule it isn't

because they're you know going crazy

back here it's it's because whenever the

capsule is torn it often actually

breaches the vitreous phase right here

if we look during development at a

neonatal or our embryonic eye we see the

the lens with the embryonic nucleus and

the fetal nucleus and then we see the

tunic of a skull osa lentes which is

contiguous with cloakers canal and this

branch of vessels and this regress is

overtime and forms a a little tuft in

adults this is a a embryonic or neonatal

lens and when we do retinopathy

prematurity exams which I don't do

anymore but I did when I ran the

neonatal intensive care unit service at

Parkland Memorial Hospital in Dallas we

can see these these lenses these blood

vessels on the lense in fact when we're

doing laser for retinopathy prematurity

we try to avoid the bigger lenses into

order to avoid causing a sub capsular

cataract and here is an example of a

incompletely regressed Bergmeister is

patella the remnant of the Clos case

canal and this is a picture from dr.

Henry Kaplan who is a famous

ophthalmologist in Louisville

specializes specialists and many things

including you be honest so here are some

some work where we're comparing an

embryonic vitreous

to a middle-aged vitreous to an aged

vitreous and one of the problems we have

with examination of the vitreous is that

the optics aren't ideal we're unable to

get the slit arm far enough over to

examine the posterior vitreous

effectively because of the optics of the

pupil and if we take the vitreous out

and then look at it with these effects

we can create what's called a Tyndall

effect and we can see that in the

e

Anatomy of lens

 what is lens then. we'll cover shape of lens with lens dimensions and terms related to that then we'll look into location of lens that way the lens is situated in the eye and its attachments then parts of the lens and lens structure start with.


what is lens so lens is a transparent a vascular refracting structure in the eye which helps in focusing light rays on the retina and as lens takes part in refraction of light lens should have some power so what is the power of lens and the answer is lens contributes about a one third to the total power of i which is about 15 to 17 diopters next is shape of lens and terms relate to that so shape of lens is spherical at birth and by convex in adults as you can see in this picture the biconvex shape of lens in which the posterior surface is more curved than the anterior surface so here if i talk about radius of curvature of these surfaces then radius of curvature is always inversely proportional to the curvature itself and as anterior surface is less curved or i can say flatter so if i draw a complete circle for this curvature then this would be radius of curvature of anterior surface which is about 10 millimeter and if i make a complete or full circle for posterior surface which is more curved than the anterior surface or i can say steeper then this would be radius of curvature of posterior surface which is about six millimeter and i think now you got it that more curved surface has lesser radius of curvature and less curved surface has more radius of curvature now there are two points to know on interior and posterior surface of lens are anterior pole and posterior pole in which anterior pole is the center of anterior surface and posterior pole is the center of posterior surface and distance between these poles is measured as thickness of lens or entire posterior diameter of the lens which is about three millimeter at birth and increase to about six millimeter in older age so we talked about shape of lens which is by convex but by convex is in cross sectional view if we see lengths from front or back of the eye then lens has a circular shape so length is a combination of two circles interiorly and posteriorly and where these two circles meet we call this as equator of the lens and in cross sectional view we say these two points as equator which is actually a complete circumference now equatorial diameter of lens is about 6.5 millimeter at birth which reaches to 9 to 10 millimeter in adults and it is generally measured in nasal to temporal dimension now how the lens is suspended at its place so the reason for that is the equator of the lens is surrounded by ciliary zonules or called suspensory ligaments which has the one end attached to the lens incomplete 360 degree circumference and the other end is attached to the ciliary processes of ciliary body now we will see location of lens so the basic location of lens is behind the iris and in front of the waitress or can say lens is in contact with aquasumer anteriorly and vitrous humor posteriorly and posteriorly the transparent twitter's gel has a shallow depression in which lens is placed that depression in vitro's gel is known as patellar fossa and there is a little space present between this particular fossa and posterior surface of lens which is known as burgers space or retro lentil space now we know that lens is situated in particular fossa but how is it attached to vitreous gel so the attachment between posterior surface of lens and anterior vitreous is in a circular fashion by a ligament known as vigor's ligament or haloid capsular and the strength of this attachment decreases with age now next is lens structure or parts of lens so histologically lens is basically composed of three structures and these structures are lens capsule anterior lens epithelium and lens fibers which will see each one by one start with lens capsule so when we say.

TARBECULER MASHWORK

we will mainly discuss about the aqueous

humor drainage system.


That is involving the tobacco meshwork so. we will discuss the anatomy of the trabecular meshwork how the equation is getting drained through this tobacco meshwork how you visualize the tobacco measure that is by means of using gonioscopy so we will learn about guneoscopy the various types of gonioscopes how to use them what are the advantages and disadvantages of each okay and how you see the angle structures when you see through the bonus scope so without much delay  so as you know the equation is produced from the ciliary process here okay enters the posterior chamber passes through the pupil and then comes to the anterior chamber fills the anterior chamber and then goes to the angle of the anterior chamber okay so here we have the important structure called as the trabecular meshwork so this will pass through this trabecular meshwork and then there are so many ways through which the equation is getting drained into the venous system so we will see the trabecular meshwork in detail now so what is the little meaning of the trabecular meshwork it is nothing but a beam or the rod which will connect two structures giving support are helping in some function so it is basically like a beam or a rod-like structure okay and this trabecular meshwork is nothing but a connective tissue core which is surrounded by the endothelium so let's discuss about the trabecular meshwork this whole color thing which is shown in this picture is a trabecular meshwork hope you got the orientation so just let me name the parts here this is the scleral spur okay extending here all over 360 degree some part of the ciliary body and then the insertion of the iris and here we have the scleral sulcus okay as the trabecular meshwork gets opposed to the scleral sulcus it converts it into a canal called as slim's canal okay in the trabecular meshwork we have three parts it's like three sheets which are opposing over the scleral sulcus to form the slam scanner the innermost one is the evasculer meshwork the second is the corneus clearer and the third is  canaliculi just go by the name so this is the uvl meshwork which is in opposition to the iris the corneas clearance okay and then the juxta which is around the slams canal so let's see each one now so this is the blue part is the uvl meshwork so this blue colored one is the uvl meshwork it is the innermost part it is in direct contact with the 8-way tumor it is attached here to the ciliary body and the root of iris to the peripheral part of the cornea okay as you can see the pores are not so regularly arranged okay and they're quite big in size it varies from 25 to 75 microns in size okay so this is about the uvl meshwork the second one that is which is shown in the red color is the corner scleral part which is attaching from the scleral spur and opposing and getting attached to the anterior wall of the scleral sulcus okay so this is the attachment of your cornea scleral meshwork so this is in direct opposition to your slums canal so it plays a major role in the drainage of the aqueous humor and as you can see the pore size is decreased compared to the evil measure and it measures about 5 to 15 microns okay and the holes become smaller as it goes near to the canals so here the inner holes are little smaller compared to the outer holes these openings are nothing but the sieve like things which help in the drainage of the aqueous humor and one more important aspect of this corn scleral meshwork is the longitudinal muscles of the ciliary body which i have explained .